Principle of 15-lipoxygenase (15-LOX) In lipid peroxidation, including the oxidative modification of LDL, is recognized as one of the crucial events implicated in the development of atherosclerosis. The enzyme 15-LOX has been shown to promote the oxidation of lipid such as LDL, which plays initial role of developing the atherosclerosis. Therefore, it is considered as a therapeutic target for the treatment of atherosclerosis-related diseases. It also stimulates some promoting atherosclerosis factors secretion (ex: chemoattractants), which can induce many monocytes adhering to endothelial cells and entering the artery wall. The monocytes inside the artery wall turn to macrophage and engulf ox-LDL, then the artery becomes thicken. In addition, ox-LDL also causes endothelialium injury. In conclusion, the product with 15-LOX inhibition activity can be used as an agent for reducing formation in cardiovascular patients. Properties of 15-LOX Inhibitor 15-LOX Inhibitor , a concentrated fermentation broth, is soymilk cultured with beneficial lactic acid bacteria and yeasts; it is manufactured by a GMP pharmaceutical plant of Microbio Co., Ltd. in Taiwan, which is also ISO9001 certified. The product formulations were prepared at a cGMP pharmaceutical formulation plant. Use and Application Information Description of the Product There is one type of preparations: capsule containing 0.5 ml of the new dietary ingredient spray dried.
Microbio is committed in the fields of cancer, cardiovascular, immune modulation, and postmenopausal care. The business segments include clinically proven botanical drugs, pharmaceutically active ing
Microbio is committed in the fields of cancer, cardiovascular, immune modulation, and postmenopausal care. The business segments include clinically proven botanical drugs, pharmaceutically active ing
Microbio is committed in the fields of cancer, cardiovascular, immune modulation, and postmenopausal care. The business segments include clinically proven botanical drugs, pharmaceutically active ing
Principle of Glutathione (GSH) Most anticancer drugs induce apoptosis directly, thus providing less opportunity for acquired drug resistance, decrease mutagenesis and reduce toxicity. In addition, in
Principle of Glutathione (GSH) Most anticancer drugs induce apoptosis directly, thus providing less opportunity for acquired drug resistance, decrease mutagenesis and reduce toxicity. In addition, in
Principle of Glutathione (GSH) Most anticancer drugs induce apoptosis directly, thus providing less opportunity for acquired drug resistance, decrease mutagenesis and reduce toxicity. In addition, in
Principle of 15-lipoxygenase (15-LOX) In lipid peroxidation, including the oxidative modification of LDL, is recognized as one of the crucial events implicated in the development of atherosclerosis.
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